021-51111890
购物车（）
sales@molnova.cn
α-(difluoromethyl)-DL-Arginine
CAS No. 69955-43-7
α-(difluoromethyl)-DL-Arginine ( —— )
产品货号. M34763 CAS No. 69955-43-7
DL-α-(Difluoromethyl)arginine 是一种有效的酶激活且不可逆的精氨酸脱羧酶抑制剂。DL-α-(Difluoromethyl)arginine 可阻断体内 E.coli 和 Pseudomonas aeruginosa 的精氨酸脱羧酶活性。
纯度: >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| 规格 | 价格/人民币 | 库存 | 数量 |
| 5MG | ￥1378 | 有现货 |
|
| 10MG | ￥2043 | 有现货 |
|
| 25MG | ￥3311 | 有现货 |
|
| 50MG | ￥4771 | 有现货 |
|
| 100MG | ￥6561 | 有现货 |
|
| 500MG | 获取报价 | 有现货 |
|
| 1G | 获取报价 | 有现货 |
|
生物学信息
-
产品名称α-(difluoromethyl)-DL-Arginine
-
注意事项本公司产品仅用于科研实验，不得用于人体或动物的临床与诊断
-
产品简述DL-α-(Difluoromethyl)arginine 是一种有效的酶激活且不可逆的精氨酸脱羧酶抑制剂。DL-α-(Difluoromethyl)arginine 可阻断体内 E.coli 和 Pseudomonas aeruginosa 的精氨酸脱羧酶活性。
-
产品描述DL-α-(Difluoromethyl)arginine is an potent, enzyme-activated and irreversible arginine decarboxylases inhibitor. DL-α-(Difluoromethyl)arginine blocks the arginine decarboxylase activity of E.coli and Pseudomonas aeruginosa in vivo.
-
体外实验——
-
体内实验——
-
同义词——
-
通路Others
-
靶点Other Targets
-
受体Others
-
研究领域——
-
适应症——
化学信息
-
CAS Number69955-43-7
-
分子量224.21
-
分子式C7H14F2N4O2
-
纯度>98% (HPLC)
-
溶解度——
-
SMILESC(CCCNC(=N)N)(C(F)F)(C(O)=O)N
-
化学全称——
运输与储存
-
储存条件(-20℃)
-
运输条件With Ice Pack
-
稳定性≥ 2 years
参考文献
1. Kallio A, et, al. DL-alpha-(Difluoromethyl)arginine: a potent enzyme-activated irreversible inhibitor of bacterial decarboxylases. Biochemistry. 1981 May 26;20(11):3163-8.?
产品手册
关联产品
-
Nogo-66 (1-40)
Peptide fragment corresponding to residues 1 - 40 of Nogo-66, the domain of the myelin protein Nogo that inhibits axonal outgrowth. Acts as a competitive antagonist at the Nogo-66 receptor (NgR); blocks Nogo-66- and CNS myelin-induced inhibition of axonal growth, but does not reduce myelin-associated glycoprotein (MAG) inhibition of neurite outgrowth in vitro. Promotes regeneration of hemisected spinal axons and locomotor recovery following spinal injury in vivo.
-
TIC10
TIC10 灭活 Akt 和 ERK，通过 Foxo3a 诱导 TRAIL，具有卓越的化合物特性：穿过血脑屏障的递送、卓越的稳定性和改进的药代动力学。
-
Halleridone
halleridone activated Nrf2-mediated HO-1 expression and inhibited Aβ1-42-overexpressed microglial BV-2 cell activation.
